‘Free radicals’ are unstable molecules within us. They have something missing in their makeup and, to compensate for this, they attack nearby healthy cells, starting a chain-reaction of destruction in which our lipids, protein and membranes are damaged, and genes altered. The damage then continues into the heart of the healthy cell where it begins to destroy DNA, the ‘blueprint’ upon which each new cell is based. Any damage to our DNA is replicated each time the cell divides, creating more damaged cells in its own likeness. This is the process by which we age, grow wrinkled and fall victim to disease as our immune systems perish under the continuous onslaught of the free radicals within us.

However, relief is now at hand in the discovery of Procyanidolic Oligormers. They are 20 TIMES more potent than Vitamin C and 50 TIMES more potent than Vitamin E in stopping free radical attacks on healthy cells.

Researchers have now combined the cell-protecting power of Procyanidolic Oligormers with the healing power of Aloe Vera, concentrated 200 times, to create Antoxida, possibly the most important medical advance since penicillin.

They double the strength of blood vessels within hours and have powerful anti-inflammatory, anti-allergy and anti-cancer properties. They neutralise the free radicals within us and are highly effective in treating a variety of medical conditions, including the following:

STROKE and HEART ATTACKS are due to a restricted inflow of blood to the organs. By inhibiting cholesterol build-up they reduces the likelihood of stroke and heart attack.

BLOOD CLOTS are another cause of strokes and heart attacks. Free radical attacks cause blood platelets to become ?sticky? and form clots. Antioxidants combat this.

The following list that shows the amount of Lutein per serving in various foods:

Directions:

1. Look through your reading glasses or bifocals.
2. Cover the left eye.
   • While looking at the centre dot from a distance of 12 inches or so, answer the following questions:
   • Can you see all four corners of the grid?
   • Are any of the lines blurry, wavy, distorted, bent, grey, or missing?

3. Repeat the previous step, this time covering the right eye.

If you note any changes in how you see the grid, call your eye doctor for evaluation.

We recommend that you keep the grid where you can use it 2-3 times a week.

Use the Amsler grid, as well as the chart on the following page, to plot your recovery progress with your action plan.

What is N-acetylcarnosine?

N-acetylcarnosine is a di-peptide, (that’s two amino acids linked together). The particularly pure form is known as n-alpha-acetylcarnosine and even acetylcarnosine. Sometimes N-acetylcarnosine is abbreviated to NAC, unfortunately that particular abbreviation is sometimes confused with N-acetylcysteine, which is not the same substance. So for the purposes of clarity on this web site, we shall continue to refer to it as N-acetylcarnosine.

What makes N-acetylcarnosine different from L-carnosine?

As an eye-drop what has N-acetylcarnosine been clinically shown to do?

N-acetylcarnosine eye-drops have been shown to help reduce, reverse and slow the occurrence of senile cataract (to be specific this was the purest form known as n-alpha-acetylcarnosine, but for simplicity we will continue to refer to it as n-acetylcarnosine).

Senile cataract is the most commonly performed surgical operation in the world today, some 26,000 people each year, in the United States alone are diagnosed with senile cataract each day. There are 1.35 million eye operations each year in the USA and 2% of them (or 27,000 individuals) develop serious complications as a result of cataract surgery.

Apart from the discomfort experienced by patients as their vision diminishes, and the possibility of being one of the surgical complications, it should not be overlooked that there is a complete lack of accommodation by the removal of the lens. At the end of the day, an artificial lens does not have the optical qualities of a natural lens.

N-acetylcarnosine eye-drops are therefore a major advancement in the control of the age-aging disorder of senile cataract.

The statistics in the human trials show that N-acetylcarnosine eye-drops applied for 6-months, (twice daily into the eye), in patients all suffering from senile cataract, had the following results:

1. 88.9% had an improvement of glare sensitivity.
2. 41.5% had an improvement of the transmissivity of the lens.
3. 90% had an improvement in visual acuity.

This is a genuine breakthrough, because before the development on N-acetylcarnosine eye-drops, the best that could be hoped for would be a slowing of the disorder. This is the very first time that it has been shown that senile cataract can be reversed without surgery.

CAN-C – A.K.A. N-acetylcarnosine

Carnosines Letter

This area is dedicated to the new clinical discoveries of N-acetylcarnosine, and how it has been shown to be effective in various aging disorders, particularly senile cataract, when used as an eye-drop.

What is N-acetylcarnosine?

N-acetylcarnosine is a di-peptide, (that’s two amino acids linked together). The particularly pure form is known as n-alpha-acetylcarnosine and even acetylcarnosine. Sometimes N-acetylcarnosine is abbreviated to NAC, unfortunately that particular abbreviation is sometimes confused with N-acetylcysteine, which is not the same substance. So for the purposes of clarity on this web site, we shall continue to refer to it as N-acetylcarnosine.

What makes N-acetylcarnosine different from L-carnosine?

As you may imagine these chemicals are related, but they are different in terms of structure.

N-acetylcarnosine C11H16N4O4

L-carnosine C9H14N4O3

As an eye-drop what has N-acetylcarnosine been clinically shown to do?

N-acetylcarnosine eye-drops have been shown to help reduce, reverse and slow the occurrence of senile cataract (to be specific this was the purest form known as n-alpha-acetylcarnosine, but for simplicity we will continue to refer to it as n-acetylcarnosine).

Senile cataract is the most commonly performed surgical operation in the world today, some 26,000 people each year, in the United States alone are diagnosed with senile cataract each day. There are 1.35 million eye operations each year in the USA and 2% of them (or 27,000 individuals) develop serious complications as a result of cataract surgery.

Apart from the discomfort experienced by patients as their vision diminishes, and the possibility of being one of the surgical complications, it should not be overlooked that there is a complete lack of accommodation by the removal of the lens. At the end of the day, an artificial lens does not have the optical qualities of a natural lens.

N-acetylcarnosine eye-drops are therefore a major advancement in the control of the age-aging disorder of senile cataract.

The statistics in the human trials show that N-acetylcarnosine eye-drops applied for 6-months, (twice daily into the eye), in patients all suffering from senile cataract, had the following results:

1. 88.9% had an improvement of glare sensitivity.
2. 41.5% had an improvement of the transmissivity of the lens.
3. 90% had an improvement in visual acuity.

This is a genuine breakthrough, because before the development on N-acetylcarnosine eye-drops, the best that could be hoped for would be a slowing of the disorder. This is the very first time that it has been shown that senile cataract can be reversed without surgery.

How does N-acetylcarnosine work in the eye?

As you may imagine these chemicals are related, but they are different.

As you can imagine this is a complex subject. But essentially, N-acetylcarnosine is a “carrier” for the di-peptide, L-carnosine into the aqueous humor of the eye, (this is the fluid area surrounding the lens). It is here, that the substance becomes most active in its ability to basically act, as a natural and comprehensive anti-oxidant.

Once N-acetylcarnosine has delivered L-carnosine safely through into the aqueous humor, the L-carnosine itself is flushed out of the eye via the canal of schlemm, and once into the bloodstream, it is broken down by carnosinase and excreted.

N-acetylcarnosine is acting as a time release version of carnosine resistant to hydrolysis with carnosinases.

Please note that L-carnosine itself, must not be used as an eye-drop.

Can L-carnosine be used in the eye?

No! L-carnosine must not be used topically in the eye.

It has been clinically shown that topical administration of pure L-carnosine to the eye does not lead to accumulation of this compound in the aqueous humor within a reasonable amount of time, or in a concentration exceeding that of the placebo-treated eye.

Exogenous L-carnosine entering the organism is not accumulated by the tissues, but is excreted or destroyed by carnosinase, a dipeptidase enzyme that is present in blood plasma.

A topical eye application of L-carnosine would release the toxic compound, histamine, (which can severely promote oxidation reactions), before it reaches the aqueous humor, and therefore could cause other eye-problems in long-term use.

Due to its relative hydrophobicity, N-acetylcarnosine penetrates through the cornea gradually, thus being able to maintain a longer active therapeutic concentration of L-carnosine directly into the aqueous humor and the lens of the treated eye. Importantly, N-acetylcarnosine is highly resistant to hydrolysis by carnosinases. Different techniques of ocular administration only with N-acetylcarnosine, have proven its efficacy for the treatment of cataracts, combined with excellent tolerability to the eye, safety, and the lack of possible side effects.

To read more technical detail about the action of N-acetylcarnosine eye-drops read on.

Cataract is caused by the nuclear matter of the elderly human lens hardening and taking on a yellowish/ brown color. Hence, once this occurs the obvious vision impairment and deterioration.

This hardening and discoloration of the lens is the result of lifelong cross-linking (or glycosylation), of the lens proteins with ascorbate. This persists in the aqueous humor at high concentrations, due to the kynurenine derivatives, and takes place due to the low availability of natural defenses in the form of anti-oxidants, (which decline with advancing age).

N-acetylcarnosine and intraocular released carnosine can effectively cleave these formed links partially, and scavenge the radicals of tryptophan/ kynurenine and ascorbate radicals. This can be measured by the intensity of fluorescence becoming decreased.

The major role of N-acetylcarnosine and L-carnosine, is their facility to act in the biological system as universal antioxidants. They possess the ability to protect cells from oxidative stress, both in the lipid phase of cellular membranes and in the aqueous environment. L-carnosine is able to reduce the content of lipid peroxidation products in the peroxidized lens fiber cells plasma membranes, and to repair their structure accordingly. It is the only known antioxidant able to protect structural proteins of the lens/ alpha-crystalline, from the free-radical induced oxidation process.

Another advantage is the capability of L-carnosine to stimulate the activity of antioxidant enzymes like SOD in the lens. SOD activity is diminished in the ageing and cataractogenesis processes.

Furthermore, there is published material about the reactivity of carnosine and its derivatives, to quench the potent free radicals, the singlet oxygen species.

Dr. Hipkiss et al., is convinced that carnosine protects protein carbonyls via the carnosylation process. In other studies, it has been indicated that carnosine has the ability to block the glycosylation process, being induced by ascorbate and transition metal ions in the aqueous humor.

It is further believed, that N-acetylcarnosine can prevent and reverse the cross-linking of the lens proteins, including crystallins induced by lipid hydroperoxides and their secondary breakdown molecular products, like aldehydes. This mechanism may be the prominent factor in reversing and preventing the lens opacification, that is related to the glycation reactions of the lens proteins.

Can N-acetylcarnosine be used orally?

No! N-acetylcarnosine must not be used orally.

Because N-acetylcarnosine is resistant to an enzyme called carnisinases, (whereas L-carnosine is not very resistant), some companies have been promoting very high oral dosages of L-carnosine to try to overcome breakdown. Therefore, some people have thought (incorrectly), that N-acetylcarnosine can be used orally instead of L-carnosine.

But, the reason that N-acetylcarnosine cannot be used orally is because it is not absorbed from the gastrointestinal tract. If N-acetylcarnosine is taken orally, then over time it can release histamine into the intestine. When used at low concentrations, this has induced food anaphylaxis, which lead to shortened lives for flies.

If you want to learn more about why N-acetylcarnosine is not absorbed from the intestine to the plasma upon its oral consumption, we offer the following published article:

High-performance liquid chromatographic determination of N-acetyl-L-carnosine in equine plasma.
Journal of Chromatography Biomedical Applications, 688 (1997) 150-154.

Until more is understood about N-acetylcarnosine and its oral use in humans, this route of administration must be avoided. However, N-acetylcarnosine has been shown to be safe and effective for humans when used as an eye-drop or as an intra-nasal spray.

How long will I have to use N-acetylcarnosine eye-drops?

N-acetylcarnosine has been shown to have measurable affects within only 1-month!

However, it is recommended that for maximum efficacy, that administration be continued for a period not less than 3-5 months. As most of the clinical trials have been measured at 3 and 6 month periods, a period of 6-months should be considered. Treatment dosages are at 1 or 2 drops, twice a day.

However, it should be considered that senile cataract is an aging disorder. It is because of low anti-oxidant and higher free radical activity, that N-acetylcarnosine may be required on a regular basis to help maintain the eye’s natural anti-oxidant defenses and therefore help to prevent a reappearance of senile cataract and other eye degenerative disorders. As a preventative measure, 1-drop into the eye once a day with frequent breaks may be a suitable on-going regime.

Will N-acetylcarnosine eye-drops be useful for other eye-disorders?

Other than senile cataract, N-acetylcarnosine may have other benefits. Although the information is not yet published, Dr. Mark Babizhayev believes that the unique N-acetylcarnosine formula with its added and synergistic lubricants, could also have benefit in the following eye-disorders:

1. Presbyopia.
2. Open-angle primary glaucoma (in combination with beta-blockers).
3. Corneal disorders.
4. Computer vision syndrome.
5. Eye strain.
6. Ocular inflammation.
7. Blurred vision.
8. Dry eye syndrome.
9. Retinal diseases.
10. Vitreous opacities and lesions.
11. Complications of diabetes mellitus and other systemic diseases.
12. Benefit for those people who wear contact lenses, particularly soft contact lenses. (Not only do the lubricants in the Can-C® n-acetylcarnosine eye-drop help to make wearing contact lenses more comfortable, but n-acetylcarnosine is also believed to reduce the build up of lactic acid in the eye, thus enabling the lens to be left safely in the eye for longer).

Bread and cereals may pose as much of a threat to children’s eyesight as reading books or staring at computer screens for too long, research suggests.

Scientists suspect the problem lies with the fact that diets high in refined starches increase insulin levels. This affects the development of the eyeball, making it abnormally long and causing short-sightedness, they suggest.

Dr Loren Cordain, from Colorado State University in Fort Collins, USA, and Professor Jennie Brand Miller, from the University of Sydney, Australia, say their theory could explain the “dramatic” increase in myopia in developed countries over the last 200 years. For example, 30 per cent of people of European descent now have myopia.

The rate of starch digestion is faster with modern processed breads and cereals, researchers explain in New Scientist magazine. In response to this rapid digestion, the levels of insulin produced by the pancreas increase, upsetting other chemical reactions. That could disturb the delicate choreography that normally co-ordinates eyeball lengthening and lens growth, leading to myopia, say researchers.

Although studies have yet to be conducted to test the theory, it is supported by some population evidence. For example; less than 1 per cent of the Inuit and Pacific Islanders had myopia early in the last century, but rates have since jumped to as high as 50 per cent.

These “overnight epidemics” are usually blamed on the rise in reading following the sudden advent of literacy and compulsory schooling in these societies.

But while reading may play a role, it fails to explain why the incidence of myopia has remained low in countries that have adopted Western lifestyles but not Western diets, says Dr Cordain. “In the islands of Vanuatu they have eight hours of compulsory schooling a day, yet the rate of myopia in these children is only 2 per cent,” he says. He believes the difference lies in the fact that the people there eat fish, yam and coconut, rather than white bread and cereal.

The starch theory is also consistent with observations that people are more likely to develop myopia if they are overweight or have adult-onset diabetes, both of which involve elevated glucose levels. The progression of myopia has also been shown to be slower in children whose protein consumption is increased.

Dr Bill Stell, of the University of Calgary, Canada, is impressed by the argument, but Professor James Mertz from the New England College of Optometry in Boston, USA, is yet to be convinced. “It’s a very surprising idea,” he says. However, the college is planning studies to test the theory in animals.

© Health Media Ltd 2003

Dry Eyes/Blepharitis Myths: Dry Eyes/Blepharitis is just a condition of eyes.

Facts: The eyes often reflect a larger problem that needs to be treated systemically. Overview, Dry Eyes: Dry eyes, often referred to as Dry Eye Syndrome, is the most frequent patient complaint to eye doctors. About 15% of the population in all groups experience varying degrees of dry eye syndromes. Like most eye conditions, Dry Eye Syndrome is often related to health conditions in the rest of the body. It is commonly associated with dryness of other mucous membranes, interior body surfaces such as joints and brittle nails. It can also be a sign of digestive imbalances (see Starch Study) or of more serious systemic autoimmune diseases, such as rheumatoid arthritis, Sjogrens syndrome or lupus erthematosus.

The proper production of tears basically takes place at three layers: * The Mucus Layer – is the closest layer to the corneal epithelium. It is produced by the conjunctival goblet cells, and is absorbed by the corneal surface glycoproteins, creating a hydrophilic corneal surface. * The Aqueous Layer – is between the Mucous and Lipid Layers, and comprises 90% of the tear film’s thickness. It is secreted by the lacrimal gland and incorporates all water-soluble components of the tear film (slightly alkaline at pH of 7.4). * The Lipid Layer – is the most superficial layer. The Meibomiam glands produce it with minor contribution from glands of Zeis and Moll. The secretion is a sebaceous material that is fluid at body temperature, and retards evaporation of the aqueous layer and lowers surface tension thereby allowing tear-film to mould itself to the eye’s surface.

* The Blink renews the tear film by delivering aqueous and lipid to tear film and sweeping away debris. The normal blink interval is every 5 seconds. Tear film is typically stable for about 10 seconds. << back to the start Symptoms: The typical symptoms include dryness, grittiness, irritation, difficulty reading for long periods of time, burning and even the seeming contradiction of excessive tearing or watering. Causes: * Any disruption in the tear production process as described in the Overview.

* Blepharitis can often cause dry eye symptoms due to inflammation of the eyelid margins, which is caused by a bacterial infection (Staphylococci). This condition can compromise the quality of the tear film causing tears to evaporate more quickly. The bacteria produce waste material that can cause a mild toxic reaction leading to chronic red, irritated eyes. Click Blepharitis for treatment.

* Computer Users tend to blink much less frequently (about 7 times per minute vs. a normal rate of around 22 times/minute). This leads to increased evaporation along with the fatigue and eyestrain associated with staring at a computer monitor. Ideally, computer users should take short breaks about every 20 minutes to reduce this factor. Also, adjusting the monitor so that it is below eye level will allow the upper lid to be positioned lower and cover more of the eye’s surface, again to reduce evaporation.

* LASIK surgery temporarily disrupts the ocular surface/lacrimal gland unit. Also, during LASIK, roughly 60-70% of the superficial nerve fibres in the cornea are severed, which impacts sensation and affects aqueous tears. With compromised sensation, the blink rate can slow to the point where the tear film breaks up before the next blink can reconstitute. This may result in mild to severe dry eye syndrome for many months after surgery. This condition usually eventually clears up.

* Diseases that may be associated with dry eyes include Rheumatoid Arthritis, Diabetes (especially when the blood sugar is up), Asthma, Thyroid disease (lower lid does not move when blinking), Lupus, and possibly Glaucoma.

* Age – Tear volume decreases as much as 60% by age 65 from that at age 18. Dry Eye Syndrome affects 75% of people over age 65.

* Hormonal changes for women can cause decreased tear production brought on by pregnancy, lactation, menstruation, and post menopause.

* Other including too much coffee drinking, smoking, wearing contact lenses, air-conditioning or heat. << back to the start Drugs can cause dry eye symptoms:

* Blood pressure medications

* Antibiotics

* Antidepressants

* Diuretics

* Over-the-counter vasoconstrictors (i.e. Visine)

* Antihistamines

* Birth control pills

* Appetite suppressants

* Ulcer medications Conventional Treatment: Artificial Tears: Some form of over-the-counter artificial tears is usually recommended. Although they may provide temporary relief, they merely palliate the symptoms. Worse, the preservatives can aggravate the condition, and can even kill corneal cells. Tears that promise to “get the red out” will reduce circulation in the eye, decrease production of the tear film, and worse, eventually make your eyes even drier.

Punctal Occlusion: Punctal occlusion is a procedure used to help dry eye patients by closing the tear drainage canals with silicone plugs, which keep most of the fluid from the surface of the eye. This may provide long-term relief. Complementary Treatment: Almost certainly, lifestyle changes will be needed to resolve the problem in the long term.

See the action plan for details.

Blurred and deteriorating vision is a common occurrence with MS. The reason is almost certainly caused when the myelin sheath develops scar tissue after being damaged during an auto-immune-system attack.

Conventional Treatment:

The existing conventional treatments that claim to work do no more than halt the deterioration.

Alternative:

Firstly take the enzyme Serrapeptase (Serrazyme). This enzyme dissolves the scar tissue that is blocking the nerve impulses. This in itself will give a remission. Then put the action plan into operation immediately.

What is diabetes?

About one person in fifty in the UK is affected by diabetes mellitus or “sugar diabetes”. As a result, the body cannot normally cope with starchy foods, sugar and other carbohydrates in the diet (or any food that elevates blood glucose levels).

Diabetes can start in childhood usually by a viral attack. In later life too many starchy foods, sugar and other carbohydrates in the diet usually cause it.

If diet is not strictly adhered to then it will cause complications that affect different parts of the body.

There are two types of diabetes mellitus – one is controlled by insulin injections, and the other by diet or tablets, but they both affect the eyes in the same way (see Starch Study).

If you have diabetes this does not necessarily mean that your sight will be affected, but there is a higher risk. If your diabetes is well controlled by diet then you are less likely to have problems, or they may be less serious. However if there are complications that affect the eyes then this can result in loss of sight.

Why regular eye tests are important

Most sight loss from diabetic retinopathy can be prevented. But it is vital that it is diagnosed early. You may not realise that there is anything wrong with your eyesight, and so regular eye checks are extremely important.
How can diabetes affect the eye?

Your eye has a lens and an aperture (opening) at the front, which adjusts to bring objects into focus on the retina at the back of the eye. The retina is made up of a delicate tissue that is sensitive to light, rather like the film in a camera.

What causes a cataract?

At the centre of the retina is the macula, which is a small area about the size of a pinhead. This is the most highly specialised part of the retina and it is vital because it enables you to see fine detail and read small print. The other parts of the retina give you side vision (peripheral vision). Filling the cavity of the eye in front of the retina is a clear jelly-like substance called the vitreous humour.

Diabetes can affect the eye in a number of ways. These usually involve the fine network of blood vessels in the retina – hence the term diabetic retinopathy.

Temporary blurring

Your vision may become blurred for a few days or weeks while your diabetes is first being controlled. This is due to the swelling of the lens of the eye and will soon clear without treatment soon after the diabetes is controlled.

Cataract

This can occur in two forms:

Diabetic retinopathy

The most serious diabetic eye condition involves the retina and is called diabetic retinopathy.

Background diabetic retinopathy

This condition is very common in people who have had diabetes for a long time. Your doctor may be able to see abnormalities in your eyes, but there is no threat to your sight.

There are two types of diabetic retinopathy that can damage your sight. Both involve the fine network of blood vessels in the retina. They are described below.

Maculopathy

This happens when the blood vessels in the retina start to leak.

Proliferative diabetic retinopathy

Sometimes diabetes can cause the blood vessels in the retina to become blocked. If this happens then new blood vessels form in the eye. This is nature’s way of trying to repair the damage so that the retina has a new blood supply.

Unfortunately these new blood vessels are weak. They are also in the wrong place – growing on the surface of the retina and into the vitreous jelly. As a result these blood vessels can bleed very easily and cause scar tissue to form in the eye. The scarring pulls and distorts the retina. When the retina is pulled out of position this is called retinal detachment.

With treatment most sight-threatening diabetic problems can be prevented if caught early enough.

Conventional treatment?

Most sight-threatening diabetic problems can be prevented by laser treatment if it is given early enough. It is important to realise however that laser treatment aims to save the sight you have – not to make it better. The laser, a beam of high intensity light, can be focused with extreme precision. So the blood vessels that are leaking fluid into the retina can be sealed.

If new blood vessels are growing, more extensive laser treatment has to be carried out. In eight out of ten cases laser treatment causes the new blood vessels to disappear. It can leave you worse off in the other 20% of cases.

Alternative Treatment?

Most sight-threatening diabetic problems can be prevented by diet, nutrition and Micro-current stimulation. If you have already had laser treatment then you may not get the recovery a well as someone who has not had laser. The critical thing is to follow the diet in the action plan.

Myths: Computers do not harm your eyes.

Facts: Computers are the number 1 cause of eyestrain.

Overview:

According to information published on November 19, 1999, “Computer vision syndrome (CVS), defined as a complex of eye and vision problems that are experienced during and related to computer use, is a repetitive strain disorder that appears to be growing rapidly, with some studies estimating that 90 percent of the 70 million U.S. workers using computers for more than 3 hours per day experience it (CVS) in some form.”

The use of computers in the western world is growing exponentially. The amount of time one spends looking at a computer screen is also increasing similarly. Humans evolved biologically as “hunters and gatherers”. Our vision developed primarily for seeing distance (98% of all humans are born farsighted). Our eye muscle systems are in their most relaxed state when we use our vision for distance objects and space. In similar fashion, our bodies were designed for movement. Maintaining a sitting posture for long periods of time is unnatural for us.

As a result, working at a computer for a long period of time without breaks can cause unnatural strain on us that can result in a condition called “computer fatigue syndrome”. Computer users have shown to have a reduced average blinking time while using computers, which, according to Japanese investigators, causes a major risk of developing transient, or short-term dry eyes.

Over a period of time, excessive computer use can have cumulative negative effects on the user including the worsening of farsightedness, nearsightedness, astigmatism, eye-focusing disorders and poor eye coordination. In addition, constant working from a set position can cause neck and shoulder stiffness, as well as stress headaches, which can then cause pain in the jaw (referred to TMJ or temporomandibular joint).

Symptoms:

-Eyestrain
-Blurred vision
-Dizziness or nausea
-Headaches
-Change in colour perception
-Increase in nearsightedness
-Red, dry or burning eyes
-Slow refocusing
-Excessive fatigue
-Neck, shoulder and back pain
-Eye-teaming problems and/or occasional double vision

Causes:

-Extending short distance focusing
-Reduced average blinking time
-Poor lighting
-Poor Posture
-Excessive glare
-Starchy Diet (see Starch Study)

Conventional Treatment:

-Aspirin
-Stronger glasses

Complementary Treatment:

The Recovery Plan is essential to alleviate this problem.

Self Help:

There are a number of simple things you can do to help protect your vision when using computers, including the following:

What is the Macula?

At the back of the eye is a layer of light sensitive tissue called the retina. The macula is found at the centre of the retina where the incoming rays of light are focused. The macula is very important and responsible for:

1) What we see straight in front of us

2) The vision needed for detailed activities such as reading and writing

3) Our ability to see colour.

What is Age-Related Macular Degeneration (AMD)?

Sometimes the delicate cells of the macula become damaged and stop working. Almost certainly it is some sort of nutritional deficiency that can happen at any age, although it tends to happen, as people get older and is referred to as age-related macular degeneration.

AMD is the most common cause of poor sight among people over 60 but while it never leads to 100% complete sight loss, it may result in only 5% peripheral or side vision will remaining undamaged. There are two different types of AMD:

You will need a good consultation with an eye doctor to identify precisely what is wrong. BUT, most Doctors do not believe anything can be done. You are going to need to take your own health in your own hands. Eyesight Action will help your eye health recovery.

What treatment is available for AMD?

Dry AMD

Although there is currently no medical treatment available for dry AMD, you can be helped to see well with nutritional supplements and stimulation of points around the eye with micro-current stimulation. Some doctors already will offer or refer you to this although the majority while not offering any treatment themselves do not.

Wet AMD

People with wet AMD can also be helped to see well with the same nutritional and micro-current stimulation program. There is also some laser treatment available for wet AMD, however it is not suitable for use in the majority of cases and can leave you permanently worse off. The laser should be used as a last resort.

Laser treatment

Laser, a device that gives an intense beam of light, can sometimes treat wet AMD. This is usually done as an outpatient and although it may cause some discomfort, is not painful. A special contact lens is put onto the eye to help focus the laser onto the macula and the heat from the laser beam then destroys the leaky blood vessels. The benefit of this treatment is that it can prevent further degeneration of the macula. However, the laser treatment can damage healthy macula cells meaning that you might lose a little sight to save a lot. Laser treatment is only useful in about 10 per cent of people with wet AMD and this always where people have reported their symptoms early.

Photodynamic therapy

A new treatment for wet AMD has recently been announced, however this treatment is still in the experimental stages and is not openly available to everyone. This new treatment is called photodynamic therapy (PDT).

With PDT a light-sensitive dye is injected into the bloodstream and is transported to the retina at the back of the eye. This chemical highlights the blood vessels that are growing abnormally. A low energy or ‘cold’ laser beam is then shone onto the macula. This makes the chemical react and destroy the leaking blood vessels without damaging the healthy tissue around the affected area in the back of the eye. More than one treatment is usually required.

The long-term effects of this treatment or its success have yet to be proved. It may only help those newly developed wet AMD, when it has been caught in the early stages.

PDT is unsuitable for long-established wet AMD and cannot restore sight that has already been damaged by macular degeneration. PDT cannot be used to treat dry AMD.

Other treatments

There are a number of other possible treatments currently being explored, such as radiation therapy, drug therapy and surgery. The development of new medical treatments takes a long time and must be rigorously tested before being made available to the public. The benefit of these types of treatments remains uncertain and you should consider the nutritional route first.

Action Plan for Recovery

Macular degeneration is now being detected in children. This may be genetic or as a result of the poor diet of the mother during pregnancy or of the child (some children only eat vegetables 3 times per week).

Whatever the cause the good news is that they respond well to nutiritional supplements and a change of diet. Whatever the medical opinion is, the fact that nutritional intervention is the most sensible option cannot be disputed.

Floaters are the name for these small dark shapes that appear before your eyes floating through your field of vision. They are clumps of protein. They look like dots, squiggles, strands or any of a hundred other shapes. Though annoying, floaters are harmless, and there are ways to prevent them through proper diet.

Warning: If you suddenly notice new spots in your vision, see your eye doctor right away to rule out serious problems. A sudden increase of floaters can be an indication of a vitreous or retinal detachment. People who are very nearsighted and/or diabetic are particularly prone to floaters and retinal tears. If you suddenly see new floaters, make an appointment with your eye doctor immediately.

The Causes of Floaters:

Floaters are usually the result of bits of cells that remain in the vitreous humor after birth and float around when the vitreous becomes more liquified later in life. It is reported that over 50% of people over 70 years of age report seeing floaters.

Myths:

Nearsightedness is a genetic condition and only gets worse.

Facts:

Nearsightedness can be improved in a majority of cases through vision therapy, nutrition, and lifestyle changes. (Longsighted also responds to the same measures).

Overview:

Myopia is defined as nearsightedness. This means you can focus better on near objects such as when reading than on distant objects. How well you see near varies depending on the amount of nearsightedness a person has. Over 1/3rd of the population are nearsighted. Interestingly, less than 20 per cent of those are born nearsighted. New studies show that the nearsightedness in these 20% was almost certainly caused by mothers eating high levels of starchy food during pregnancy.

Symptoms:

Difficulty seeing details in the distance clearly without the use of glasses.

Causes:

The causes of myopia can vary from person to person, but below are some of the variables that may affect one’s near vision:

Conventional Treatment:

Glasses with prescriptions that tend to become stronger every one to two years.

Alternative Treatment:

There are a number of steps shown in the Action Plan.

Overview:

Conjunctivitis is the most common eye disease in the Western hemisphere. Because of its exposed position, the conjunctiva comes into contact with more micro organisms than any other membrane. Bacteria, such as pneumococcus, staphylococcus or streptococcus, cause some cases although allergies and some viruses can also cause conjunctivitis. A virus called adenovirus causes other cases. This virus can be spread in many ways including swimming pools, wet towels, etc. The infection usually starts in one eye and is spread to the other quite readily. It usually results in red, irritated eyes and disappears in three days to a week. If the symptoms linger, go to the eye doctor. The infection can enter the cornea and create little clouded areas that may affect vision.

Symptoms:

Causes:

Drugs can cause allergic conjunctivitis:

Antibiotics – when antibiotics are given for eye problems topically they may have the side effect of causing an allergic conjunctivitis (red eye). Systemic antibiotics taken orally, intramuscularly, or intravenously to help with bacterial infections may cause some visual symptoms. For example:

1. Synthetic penicillins (amoxicillin and ampicillin) – a person taking these may experience some mild redness of the eyes, itching and dry eyes. In rare cases they have been shown to cause haemorrhages of the blood vessels in the conjunctiva and in the retina

2. Tetracycline- similar to the above plus light sensitivity and blurred vision

3. Sulfonamides – many people are allergic to “sulfa drugs”. This can cause blurred vision, light sensitivity and haemorrhages in the eye.

Note Whenever taking antibiotics make sure you take probiotics such as acidophilus or bifidus and vitamin C to help ward off some of the side effects of the antibiotics.

Conventional Treatment:

Conventional medicine usually prescribes sulfa-based eyedrops once other potential problems have been ruled out. These usually work within three days. If not, broad-spectrum antibiotics are prescribed. In the case of a virus, not much can be done. An old time traditional treatment includes a 1% silver nitrate or 5% povidone/iodine solution – this will help eliminate the virus-infected cells by exfoliating the surface of the eye, as well as killing the virus itself.

Alternative Treatment:

Conjunctivitis is best treated with nutrition and natural remedies (See Action Plan).

DISSOLVE CATARACTS WITH EYE DROPS? CLICK HERE!

What is a cataract?

A cataract is a clouding of part of your eye called the lens. Your vision becomes blurred or dim because light cannot pass through the clouded lens to the back of the eye.

The lens

dissolved

The lens is a transparent body behind the iris, the coloured part of the eye. The lens bends light rays so that they give a clear image to the back of the eye – the retina. As the lens is elastic, it will change shape, getting fatter for close objects and thinner for distant objects. The diagram below shows the main parts of the eye.

What causes a cataract?

Cataracts can form at any age, but most often they develop as people get older due to lack of antioxidants in the diet that counteract the free radical damage that is particularly prevalent in the eye. In others and younger people they can result from an injury, certain drugs, long-standing inflammation or illnesses such as diabetes which is subject to extra free radical damage as a result of high glucose levels (see Starch Study).

Some symptoms

Congenital Cataract?

A congenital cataract is an opacity (cloudiness) in the lens of the eye that is present at, or develops shortly after, birth.

The human lens is located behind the pupil and has an important function. It focuses rays of light into the retina to allow image formation. As the lens is able to change is shape, it can focus objects at different distances. Its cells are arranged so that it is transparent, like glass or water. When this arrangement is disturbed in any way, the transparency is lost and an opacity results. This results in blurring and blocking of the retinal image.

In the case of a newborn infant, a cataract causes the immature visual system to be deprived of the stimulation needed for normal development. If left untreated, permanent visual loss may occur. It is thought that starchy foods eaten in high levels during the pregnancy or given to the infant in the first 24 months may be the cause of the problem.

Inherited Congenital Cataracts

Approximately 0.03% of newborns have some form of congenital cataract. Most are not associated with additional development problems. Around one fifth of these have a family history of congenital cataract but in up to half of all cases there is no family history. Congenital cataracts at present account for the largest proportion of partially sighted and blind registered children in the UK.

What can be done to help?

Glaucoma is defined as a collection of diseases that causes optic nerve damage.

Symptoms:

Causes:

Nutritional Intervention:

Damage to the optic nerve can still happen with eye drop controlled pressures. Nutritional support, supplementation and lifestyle changes can dramatically improve the condition of your eyes.

As will most childhood problems a squint is almost certainly caused by either a nutritional deficiency during the development of the baby in the womb or damage caused by eating high levels of starchy foods during pregnancy (see Starch Study). This may also be contributed to by the infant being given starchy food in its formative years.

Conventional Treatment:

Optical correction is usually successful

Alternative Treatment:

The Action Plan can be successfully used on its own or along side the optical corrective work.

Which eye diseases are genetic?

Retinitis Pigmentosa is the most well known genetic eye disease. Many cases of severe eye disease in children such as microphthalmos (small eye), cataract, glaucoma and retinoblastoma, an eye tumour in childhood, are caused by genetic defects.

Many genetic diseases which affect several body systems also affect the eye e.g. Marfan’s syndrome, neurofibromatosis.

It is increasingly recognised that many adult eye diseases such as cataract, glaucoma and diabetes are modified by a person’s genetic constitution but it may be that diet is an important factor (See Starch Study).

Orthodox treatment:

There are some aspects of most genetic eye diseases that can be treated, but as yet treatment to put right the genetic problem itself is not available for these conditions. There is a great deal of research being done into genetic disorders, and it is reasonable to expect advances in treatment in future years.

Alternative treatment:

The doctors in the USA who pioneered the alternative treatment for Macular Degeneration also found that Retinitis Pigmentosa also responded. Quite why this is if the problem is genetic ‘in the eye’ is baffling. If the problem is lack of nutritional uptake in the digestive tract (genetic) or a problem absorbing the nutrients in the eye then it may explain ‘why’. The simple fact is it does respond at any age and the average person could not care less why it works.

We are always researching new treatment regimes and are able to give guidance in many cases.

If you have an eye disease not covered on this site, simply e-mail us with your details and we will contact you to discuss your situation.

All correspondence is dealt with in strictest confidence.

All of the listed factors need to be in place as they all work interactively or as co-factors to each other and any deficiency will result in a malfunction. For example, there are over 80 enzymes at work in your body that need zinc as a co-factor.

Only the uninformed would claim that these essential nutrients are in the average UK diet (even if your ageing digestive system could absorb them). Are you absorbing your nutrition? I will try to explain what may going wrong with your eyes as simply as possible.

You need many specific nutrients to help counteract an essential part of your body’s energy creation called, free radicals. Free radicals are partly caused by sunlight acting upon our eyes, from cellular combustion when we create energy and oxidation from oxygen activities in our body. We should have two specific antioxidants in our eyes, Lutein (lu-teen) and Zeaxanthin (zee-a-zan-than). They help to counter this free radical activity (see studies). A substance called ATP; created by our cell to provide all of our energy for life and cellular cleaning and regeneration is a third major help. We can conclude therefore that it is necessary to have these nutrients in, abundance, in our eyes.

Why do we not have them? Let us look at the antioxidants Lutein and Zeaxanthin. These are found in abundance in dark leafy green vegetables. There are five reasons why you may be lacking these nutrients in the eye:

ATP is a nutrient produced in your own cells for all of your energy needs. Your body needs the equivalent of your own body weight of ATP, every day, to supply its energy needs. To produce this, your cells (as well as essential fatty acids) need to absorb Oxygen and Glucose in a ratio of 80% to 20% for the huge amount of ATP needed. This ATP is essential as discussed earlier not only for your body’s physical energy but for cell cleaning and cell regeneration needs. Unfortunate this process causes a damaging, combustive, free radical activity (oxidation). These free radicals damage the healthy cells as a result and can only be stopped by eating a good amount of fruit and vegetables with their powerful supply of antioxidants. The antioxidants Lutein and Zeaxanthin are the specific antioxidants that alleviate this free radical activity in the eyes and are therefore vital for the healthy aging of your eyes and the rest of your body.

Premature aging and degeneration is believed by many researchers to be caused in part by an ultimate deficiency of ATP as well as free radicals. Too much glucose (from eating starchy foods, sugar, potatoes) and foods that easily covert to glucose, and a deficiency of oxygen from poor breathing, or insufficient aerobic exercising can upset this delicate 80% Oxygen:20% Glucose balance, thus causing an ATP deficiency. When this happens the cell reduces its output of ATP and produces a toxic substance called Lactic Acid.

The ATP deficiency will result in you suffering from an energy loss which may cause you not to exercise enough. To make matters worse you may also mistakenly take in more sugars and carbohydrates to try to give you more energy thereby poisoning your cells regeneration even more. This by a process of glucose damage to your cells is known as Glycation. Even worse is that glucose damage to proteins known as ?glycated proteins? produce 50 times more free radicals than non glycated proteins and research indicates Glycation may also be prime factor in brain diseases. A lack of antioxidants, ATP and the extra work caused by Glycation will inhibit your cells ability to regenerate thereby causing overall premature aging and DAMAGE TO YOUR EYES.

Anti-Aging Theory – There are good studies on animals in the laboratory which show that by greatly reducing calories (the biggest culprit is grains, cereal, potatoes and sugars) the life of animals on low calorie: vegetables, nuts and seeds, doubled, compared to those eating high calorie foods. The animals eating low calories foods did not suffer from age related degenerative diseases as suffered by the high calories group. Do you need any other motivation for cutting back on grains, cereals, sugars and potatoes? – See the Glycemic Index of food for balanced blood sugar foods.

Directions:

If you note any changes in how you see the grid, call your eye doctor for evaluation.

We recommend you keep the grid where you can use it 2-3 times a week.

Amsler Grid: Test Your Eyesight

MicroCurrent Stimulation Stimulates ATP and cellular regeneration and has been shown in studies to improve the overall success.Recommended use: Stimulate Appropriate MicroCurrent Points.  Treatments per day for the first 2-4 weeks and once per day thereafter.

Essential Liquid Taurine offers better absorption than capsules. Studies have shown Taurine to reduce the oxidative damage caused by sunlight to the eyes. It helps stimulate the body’s ability to clean up waste by-products that accumulate in the retina.Recommended dosage: 8 sprays per day for the first month, and then 2 per day thereafter.

Essential EFAs in liquid form must contain at least 480mg of DHA and 720mg of EPA per teaspoon. This helps improve circulation, integrity of blood vessels, brain function, flexibility and permeability of cell membranes. It also helps protect the retina’s photoreceptor cells.Recommended dosage: 2 teaspoon per day.

Essential 91 Vitamins and Minerals are liquid (3 times more absorbent than tablets, ensuring better utilisation by the body) and replaces your current vitamin/mineral formula. This includes the critical minerals selenium and chromium, as well as many others that will help your recovery.Recommended dosage: ½oz with breakfast and the same with your evening meal

Alpha Lipoic Acid is the only antioxidant that is both fat and water-soluble. Studies have shown that it is able to repair oxydative and free radical damage and its protective effect extends to nearly every cell in the body. It has also been found to enhance the action of Glutathione, vitamins E and C.Recommended dosage: 100mg 3 times per day.

MSM Eye Drops are recommended for all eye conditions and are an inexpensive self treatment

N-Acetyl Carnosine Drops - Stimulates Glutathione directly in the eye and helps dissolve cataracts, and is crucial in stopping free radical damage. Studies have shown that all lenses with cataracts contain approximately 1/5th the amount of Glutathione as compared to healthy lenses. Recommended dosage: 1 or 2 drops in each eye 1-4 times a day

N-Acetyl Cysteine Capsules - Stimulates Glutathione and offers protection against cataract formation, and is crucial in stopping free radical damage. Studies have shown that all lenses with cataracts contain approximately 1/5th the amount of Glutathione as compared to healthy lenses. Recommended dosage: 4 caps x 500mg per day.

MicroCurrent Stimulation Stimulates ATP and cellular regeneration and has been shown in studies to improve the overall success. Recommended use: Treatments per day for the first 2-4 weeks and once per day thereafter.

Essential 91 Vitamins and Minerals are liquid (3 times more absorbent than tablets, ensuring better utilisation by the body) and replaces your current vitamin/mineral formula. This includes the critical minerals selenium and chromium, as well as many others that will help your recovery. Recommended dosage: ½oz with breakfast and the same with your evening meal

Vitamin C, Plant Sourced is specifically to help reduce cataracts. Researchers report that a “significant interaction” was observed between age, vitamin C intake and the prevalence of cataracts. Recommended dosage: 3 x 1.3grams capsules per day (1 with each meal).

Recommended dosage: 8 sprays per day for the first month, and then 2 per day thereafter.

N.B. The above plan, combined with the diet change shown in section 4, can reverse the effects of Type 2 Diabetes (age related).

Pancreas Support Spray contains two important trace minerals, Vanadium and Chromium, plus the herb Gymnema Sylvestre, which studies have all shown to be effective with helping diabetes. Note that you should monitor your need for insulin or tablets, as these nutrients may lower or replace the need for these drugs.

Recommended dosage: 6 sprays per day for the first two months; then 3 sprays per day for the next two months; and lastly, 1 spray per day as a preventative.

Lutein Nutritional Sublingual Spray is a liposomal spray that contains high levels of the key carotenoids Lutein and Zeaxanthin, together with 22 vitamin and minerals identified in a major study as being essential for eye health. They have been shown to protect the retina against sunlight and oxidative damage by free radicals, strengthen the blood vessels and improve blood flow to the eye. Sprayed under the tongue, it is the most effective way to deliver these nutrients to the eye in a few minutes.

Recommended dosage: 4 sprays 3 times per day for the first 3 bottles then reduce to 2 sprays 3 times per day.

Recommended dosage: Stimulate appropriate MicroCurrent points. Treatments per day for the first 2-4 weeks, and once per day thereafter.

Essential Fatty Acids in liquid form must contain at least 480 mg of DHA and 720 mg of EPA per teaspoon. This helps improve circulation, integrity of blood vessels, brain function, flexibility, and permeability of cell membranes. It also helps protect the retina’s photoreceptor cells.

Recommended dosage: 1 teaspoon per day.

Alpha Lipoic Acid is the only antioxidant that is both fat and water-soluble. Studies have shown that patients with diabetes have increased cellular uptake and burning of glucose by approximately 50%. It has also been found to enhance the action of Glutathione and vitamins E and C.

Recommended dosage:100 mg 3 times per day.

MSM+Silver Water Drops are recommended for all eye conditions and are an inexpensive self treatment.

Massager Pin Hole Glasses can improve blood flow, help eyestrain and headaches caused by computer work, Long Sightedness, Short Sightedness and Presbyopia. Massage Glasses can relax tired eyes, build-up the flexibility of your eyes and can in some cases reduce your current prescription.Recommended use: 5-10 minutes twice per day.

Vitis Vinifera A herb containing flavenoids that in a study showed 85% of patients had a significant improvement and 40% remarkable improvements. (Ann Oft Clin Ocul, 1988; 114: 85-93).Recommended dosage: 1-3 drops per day as needed.Bates Method – A method of exercising and improving sight developed by William Bates.

Essential 91 Vitamins and Minerals are liquid (allowing for the maximum absorption and utilisation by the body) and would replace your current vitamin/Mineral formula. This includes the critical minerals selenium and chromium, as well as many others that will help your recovery.Recommended dosage: 1/2oz with breakfast and evening meal.

Highly Recommended Antioxidant Formula is specifically to help improve the integrity of the blood vessels in the eye, which is particularly important in those with Wet Macular. This formula has been shown to double the strength of blood vessel walls with hours. It contains: Vitamin ‘C’, Aloe Vera Concentrate, L-Cysteine, Vitamin ‘E’, Grape seed extract (95% proanthocyanidins), grape skin extract, Zinc, Vitamin ‘A’ as Beta Carotene, Selenium.Recommended dosage: 6 per day (2 before each meal) for the first month and then 3 per day thereafter.

MSM Eye Drops are recommended for all eye conditions and are an inexpensive self treatment.

Essential Microcurrent Stimulation has been used for the past 10 years to treat Macular Degeneration and other eye diseases. The initial results obtained by individual Doctors in the USA indicate that 70% of patients with the dry or wet form of macular degeneration will have a significant improvement of vision.

Recommended dosage: 4 sprays 3 times per day for the first 3 bottles then reduce to 2 sprays 3 times per day.

VIVA-Eye adds vitamin A directly to the eye. This acts as a lubricant, preventing irritation and dryness caused by insufficient blinking, dry air (air conditioning), and possible free radicals radiated from the computer screen.

Recommended dosage: 1-3 drops per day as needed.

Massager Pin Hole Glasses can improve blood flow and relieve eyestrain and headaches caused by computer work, farsightedness, nearsightedness and Presbyopia. Pinhole Massager Glasses can relax tired eyes, build up the flexibility of your eyes, and can, in some cases, reduce your current prescription.

Recommended use: 5-10 minutes twice per day.

MSM+Silver Water Drops are recommended for all eye conditions and are an inexpensive self treatment.

MicroCurrent Stimulation uses a gentle cotton bud probe to safely apply a tiny electrical impulse to the acupressure points around the eyes. Stimulates ATP and cellular regeneration and has been shown in studies to improve the overall success.

Recommended use: Stimulate appropriate MicroCurrent points. Treatments per day for the first 2-4 weeks, and once per day thereafter.

Recommended dosage: 4 sprays 3 times per day for the first 3 bottles then reduce to 2 sprays 3 times per day.

MicroCurrent Stimulation uses a gentle cotton bud probe to safely apply a tiny electrical impulse to the acupressure points around the eyes. Stimulates ATP and cellular regeneration and has been shown in studies to improve the overall success. Recommended use: Stimulate appropriate MicroCurrent points.

Treatments per day for the first 2-4 weeks, and once per day thereafter.

Vitis Vinifera A herb containing flavenoids that in a study showed 85% of patients had a significant improvement and 40% remarkable improvements. (Ann Oft Clin Ocul, 1988; 114: 85-93).

Recommended dosage: 1-3 drops per day as needed.

Massager Pin Hole Glasses can improve blood flow and relieve eyestrain and headaches caused by computer work, farsightedness, nearsightedness and Presbyopia. Pinhole Massager Glasses can relax tired eyes, build up the flexibility of your eyes, and can, in some cases, reduce your current prescription.

Recommended use: 5-10 minutes twice per day.

Bates Method - A method of exercising and improving sight developed by William Bates can be found in many books in the health stores and book shops (now on DVD – call helpline 01606 871203).

MSM+Silver Water Drops are recommended for all eye conditions and are an inexpensive self treatment.

Lutein Nutritional Sublingual Spray is a liposomal spray that contains high levels of the key carotenoids Lutein and Zeaxanthin, together with 22 vitamin and minerals identified in a major study as being essential for eye health. They have been shown to protect the retina against sunlight and oxidative damage by free radicals, strengthen the blood vessels and improve blood flow to the eye. Sprayed under the tongue, it is the most effective way to deliver these nutrients to the eye in a few minutes.

Recommended dosage: 4 sprays 3 times per day for the first 3 bottles then reduce to 2 sprays 3 times per day.

VIVA-Eye adds vitamin ‘A’ directly to the eye. This acts as a lubricant, preventing irritation and dryness caused by insufficient blinking, dry air (air conditioning) and possible free radicals radiated from the computer screen.

Recommended dosage: 1-3 drops per day as needed.em>

Recommended dosage: <em½ dropper full 2 times per day on an empty stomach. Do not take as eye drops.

Recommended dosage: 6-8 sprays per day for the first two months; then 3 sprays per day for the next two months; and lastly, 1 spray per day as a preventative.

MicroCurrent Stimulation uses a gentle cotton bud probe to safely apply a tiny electrical impulse to the acupressure points around the eyes. Stimulates ATP and cellular regeneration and has been shown in studies to improve the overall success.

Recommended use: Stimulate Appropriate MicroCurrent Points. 2 Treatments per day for the first 2-4 weeks and once per day thereafter.

Alpha Lipoic Acid is the only antioxidant that is both fat and water-soluble. Studies have shown that patients with glaucoma have benefited with enhanced color vision and generalized visual sensitivity. It has also been found to reduce diabetic cataract formation and enhance the action of Glutathione and vitamins E and C.

Recommended dosage: 100mg 3 times per day.

Essential Fatty Acids in liquid form must contain at least 480 mg of DHA and 720 mg of EPA per teaspoon. This helps improve circulation, integrity of blood vessels, brain function, flexibility, and permeability of cell membranes. It also helps protect the retina’s photoreceptor cells.

Recommended dosage: 2 teaspoon per day.

2 teaspoons per day.

Essential Liquid Vitamins and Minerals are 3 times more absorbent than tablets, ensuring better utilization by the body, and replaces your current vitamin/mineral formula. This includes the critical minerals selenium and chromium, as well as many others that will help your recovery.

Recommended dosage: ½ oz with breakfast and with your evening meal.

Highly Recommended Antioxidant Formula - - Astaxanthin, an extremely powerful antioxidant that is particularly recommended for preventing damage to the retina caused by strong sunlight. Studies show that it is many times more effective than Vitamin E and Beta-Carotene.

Recommended dosage: 3 x 5mg Capsules per Day.

MSM+Silver Water Drops are recommended for all eye conditions and are an inexpensive self treatment.

By the time someone is diagnosed with AMD, the disease has been developing for 20 years or more, says Dr. Steve Pratt, an ophthalmologist and chair of the Lutein Information Bureau. The good news is that, “It’s never too late and it’s never too early to start consuming Lutein for better ocular health,” he adds.

What is Lutein? Lutein (pronounced LOO-teen) is a carotenoid found in vegetables and fruits. Lutein acts as an antioxidant, protecting cells against the damaging effects of free radicals. Lutein is not made in the body. Lutein must be obtained from food or vitamin supplements. Lutein is found in large amounts in green, leafy vegetables such as spinach.

Lutein and zeaxanthin – another antioxidant found in vegetables – are the only carotenoid pigments found in the macular, observes Pratt. This may explain why Lutein works as an antioxidant in the eye to help protect it from sun damage. “Lutein and zeaxanthin probably act as a natural filter for blue light, which is very toxic to the retina, he notes.

Ongoing research also indicates that Lutein may inhibit the development of cataracts a – disease in which more than half of all Americans suffer, according to the LIB. Harvard researchers found that among the 77,000 women who participated in its Nurses’ Health Study, those with the highest intake of foods rich in Lutein and zeaxanthin were 22 percent less likely to develop cataracts than women with the lowest intake.

“Think spinach and kale,” says Pratt, with regard to the best sources of Lutein. To promote eye health, the LIB recommends the consumption of at least 42 milligrams of Lutein each week.

Not eating or absorbing your nutrition?

Doctors in the USA have been achieving amazing results with a sublingual spray containing Lutein that absorbs up to 10 times better than tablets and can be measured in the Macula within 10 minutes in all users. The brand name of this amazing spray is New Focus.

1. Stop Smoking.
2. Wear eye shades in the sun
3. Drink 10 glasses – pint of water per day.
4. Foods to exclude from your diet: grains and cereals (bread, pastry, biscuits etc.) and root vegetables (potatoes, parsnips etc) for 3-6 months. Foods to include: Vegetables – 8 portions EVERY day (kale, Spinach etc and as many different colours as possible, Fruit, Nuts, Seeds, Beans, Eggs, Fish (different fish 4-5 times per week).
5. Exercise everyday to get oxygen around your body and get your lungs working and your heart beating. Walking, running on the spot rebounding on a mini-trampoline are best.
6. Take enzymes and all other nutritional supplements that you can afford that are know to be helpful to your eyes.
7. Consider a Herbal Cleanse and replenish your Probiotic bacteria (acidophilus etc.) regularly.
8. Ask your Doctor to refer you to a Homeopathic Doctor.
9. If you have blocked arteries ask about Chelation Enzymes and Herbs to clear them without waiting for surgery.
10. Get serious about good health because your quality of life is at stake.

There are many factors related to the condition known as of macular degeneration. The most common cause is the circulation problems that increase as we age. The macula has a very high requirement for proper nutritional elements, oxygen, and the elimination of waste products in order to function properly. Circulation problems reduce the supply of oxygen and nutrients to the macula and will cause a dysfunction in the macula and, ultimately, degeneration. Electronic Stimulation is a therapy in which a weak electric current is used to stimulate the retina and the diseased macula to restore sight though points known as acupuncture points.

Dr. Merill Allen and Dr. Leland Michael published their preliminary study in 1993, on the rate of development of ARMD in people using nutritional supplements and simultaneous therapy with electronic stimulation. Dr. John Jarding reported his results in 1997 after treating thirty-five macular degeneration patients with a controlled electronic stimulation applied to eight points around the eye – all thirty-five patients reported an improvement in their vision. The Macular Degeneration Foundation in the USA has begun a national clinical trial to look at the effects of electronic stimulation and ARMD.

An Exciting New Development

An electronic device, researched over 12 years by a leading British Doctor, is designed to provide all the benefits of MicroCurrent Stimulation as well as Electronic Acupuncture (without needles). MicroCurrent Stimulation is used by health professionals in many hospitals and is designed to be used safely and effectively by the public for self-treatment.

THIS DEVICE WORKS IN TWO WAYS:

1. With the help of it’s unique searching system, enables you to quickly become expert at precisely locating therapy points

2. The device then applies electrical pulses to these points enabling you to treat any condition that acupuncture can help.

It is safe – drug-free – with no side effects, and is easy to use.

You will know you are on the correct point when you FEEL a distinct sensation. Mild electric pulses then stimulate these points.

THE EFFECTS OF THIS THERAPY ARE:

This enables you to become expert almost immediately locating and treating the exact points.

The recommended device is sold as HealthPoint in the UK and HealthTouch in the USA.

History of Micro Current Stimulation – by Larry Ratliff, President & CEO Dove Alliance USA Inc.

Back in the late 1960’s Dr. Julian Kenyon, a UK surgeon and Master Acupuncturist, had a vision of putting the healing power of acupuncture into the hands of the lay person – and as a consequence improving the effectiveness of acupuncture.

After twelve years of research, the result was the EPS. As the first micro current device of its kind, the EPS incorporated a locating system that allowed the user to locate acupuncture points with 100% accuracy and then stimulate that point with a very minute electronic pulse. When coupled with traditional acupuncture protocols the results were remarkable and repeatable.

Dr. Kenyon’s research was founded on the works of Robert Becker and others that had been researching the electrical properties of the human body. In the course of his study, Dr. Kenyon recognised an amazing connection between this western research and eastern medicine. Western research had identified specific channels of electro-magnetic energy that correspond exactly to the eastern description of meridians. Also small points exist, in exactly the same location as described acupuncture points, which are 10-40 times more conductive than the surrounding skin. When stimulated electrically these points demonstrate a direct correlation to the electro-magnetic fields in the meridians. The voltages in the meridians can be readily measured and change in relation to health or lack of health. Although the western researchers were not aware of it, they had confirmed the theory of eastern medicine.

Now Dr. Kenyon had a tool to work with and a method of cause and effect evaluation. From this beginning Dr. Kenyon went on to refine and define the most effective electrical properties and procedures to use when treating the body. It was found that bi-polar micro currents, in a range of less than 50 micro-amps, having a square waveform and frequency of 10 hertz would trigger the body’s natural healing system and promote tissue regeneration.

The current generation of the EPS is called HealthPoint or HealthTouch and has been successfully used in clinics and by lay people around the world for over 20 years. During the same period of time, it had been discovered that stimulating the body with currents in the milli-amp range could provide relief from some types of pain. As a result, western research was moving forward on devices having much higher output currents. Based on the “gate theory” of pain control, an electrical current is applied to the nervous system and interferes with the transfer of information from one nerve cell to another, preventing a signal from reaching the brain. The pain is there, the condition is there but the body does not acknowledge it. These high current devices are termed TENS devices and are widely used today for the treatment of many painful conditions.

Continued research has brought information leading to improvements in the hardware and techniques of TENS treatment. This refinement has broadened the field of use of TENS stimulation and, interestingly, reduced the usable output currents down into the micro current range. One of these researchers is Dr. Joel Rossen, an acupuncturist and veterinarian, who had a desire to improve the treatment of animals and had started building his own TENS devices.

As always, necessity is the mother of invention. By 1976 all of the ingredients for a breakthrough in Macular Degeneration treatment were in place, but existed separately and apparently unrelated in an arena that was spurned by leading researchers:

1. Dr. Kenyon had joined micro-current stimulation with acupuncture.

2. TENS research had brought outputs down into the area of micro-currents.

3. Advances in nutrition research were bringing the focus from corrective medicine to preventive lifestyle and good health practices.

Then, in 1979, Grace Halloran entered the picture. Having a great desire to cure her eye problems, an unlikely chain of events transpired to bring about a miracle cure.
Grace had hereditary Retinitis Pigmentosa (RP) and at the time was working for a football team as a consultant specialising in alternative treatment of injuries. Her friend, a physiotherapist, introduced her to Dr. Joel Rossen who was conducting training classes on the use of his TENS devices. Grace worked with Dr. Rossen for several years refining the unit design. Then a successful treatment for a serious injury to her son prompted her to try the micro-current device on her own eye condition – with some success. Grace continued to refine the treatment procedure, founded the Integrated Visual Healing program and has been spreading the word ever since.

In 1985, while presenting seminars, Grace came into contact with Dr. Leland Michael and Dr. John Jarding, who conducted a 25 patient study. Then in 1992 – 1999 came doctors Wallace, Miller, Khouri, Chee, Kondrot and Nagel. The word is spreading, Grace is still conducting seminars.

Studies.

Early studies were simply clinical trials where the treatment was administered and results recorded. This started with Grace Halloran and continues today with every doctor that provides this treatment protocol. All reports are very consistent in results and safety.

In 1993 Dr. Michael published a paper covering treatment of 25 ARMD patients over a seven-year period. (Dr. Michael died shortly after publication and his practice was taken over by Dr. John Jarding). Dr. Jarding then conducted an investigative trial over a period of seven years, concluding in 2000. Results are not yet published but are reported to be consistent with previous findings.

In 1998 Dr. Miller published a statistical analysis of ARMD patients treated with the microcurrent protocol.

Dr. Rossen and the Macular Degeneration Foundation are currently conducting an FDA study.

The Doctors.

1976 – Dr. Julian Kenyon, 1979 – Grace Halloran, 1979 – Dr. Joel Rossen, 1985 – Dr.
Leland Michael, 1985 – Dr. John Jarding, 1992 – Dr. Jerry Wallace, 1997 – Dr. Harry Miller, 1998 – Dr. George Khouri, 1998 – Dr. Percival Chee, 1998 – Dr. Edward Kondrot, 1999 – Dr. James Nagel

With the knowledge we now have – there is hope! Using a combination of nutrition and micro current stimulation, a 60% to 80% improvement in visual acuity on 80% of the patients treated and stoppage of progression has been consistently achieved, with no adverse side effects.

We can actually reverse the vision loss in the majority of cases and stop the progression of the eye disease in the rest. Research goes on and progress is being made. Reports continue to come in about amazing successes and discoveries.

Our Mission…

…is eyesight recovery or protection of any remaining eyesight and vision by guiding people along the Natural Health path with recommendations and actions based on peer review studies, professional’s and member’s experiences.

Health of the eyes is dependant on the health of one’s entire body. To recover and preserve vision we provide lifestyle, diet, prevention and nutritional recommendations.

Contact us

Macular Degeneration

Glaucoma

Diabetic Eye Problems

Cataracts

Retinitis Pigmentosa

Alpha Lipoic Acid

Press Release – Lutein-rich food studies

1993 – Data suggested a reduced risk of neovascular AMD in persons with higher levels of serum Carotenoids. (11)

1994 – Dr. Seddon found that eating foods high in Lutein led to a reduced risk for macular degeneration. (2)

1995 – Dr. Yeum reported Lutein and Zeaxanthin as the only two Carotenoid antioxidants found in the lens and retina. (1)

1997 – Studies were published showing a 30% reduced macular pigment density with AMD and cataracts. A correlation was found between reduced macular pigment density and increased lens density (cataracts) in seniors. (12)(13) With dietary and/or supplemental Lutein, macular pigment density can be increased. (14)

Latest Lutein Study – May 2002

JOURNAL REFERENCES

1. KJ Yeum et al. 1995 Investigative Ophthalmology & Visual Science 36:2756-61.
2. JM Seddon et al. 1994 Journal of the American Medical Association 272:1420.
3. F Khachik et al. 1995 Journal of Cellular Biochemistry 22:236-46.
4. LH Zhang et al. 1991 Carcinogenesis 12:2109-14.
5. DM Snodderly 1995 American Journal of Clinical Nutrition 62S:1448S-61S.
6. Chopra et al. 1994 Proc Nutritional Society 53:18A.
7. Goulient et al. 1997 Arterioscleroses Thrombosis Vascular Biology 17:4:786-96.
8. LE Marchand et al. 1995 International Journal of Cancer 63:18-23.
9. Eye Disease Control Study Group 1992 Archives of Ophthalmology 110:1701-8.
10. Hankinson et al. 1992 British Medical Journal 305:335-9.
11. Eye Disease Control Study Group 1993 Archives of Ophthalmology 111:104-9.
12. JT Landrum et al. 1997 Advances in Pharmacology 38:537-56.11
13. BR Hammond, Jr. et al. 1997 Optometry and Vision Science 74:7:499-504.
14. BR Hammond, Jr. et al.1997 Investigative Ophthalmology & Visual Science 38:1795-1801.

ATLANTA, Feb. 25 /PRNewswire/ – Dietary modification with a Lutein-rich food can reverse the damaging effects of age-related macular degeneration (ARMD), the leading cause of blindness in people over age 65 in the United States, according to a report presented today by Stuart Richer, O.D. at the Southern Council of Optometrists 1999 annual meeting. Until this point, research only indicated that diet plays an important role in reducing disease risk.

“Patients demonstrated positive effects in visual function in one or both eyes with the simple addition of rich-rich foods” reported Richer, chief of the Optometry Section, DVA Medical Centre, North Chicago, IL. “This research supports the hypothesis that Lutein is associated with building macular pigment, a key indicator of ARMD risk and pathology.”

Richer supplemented the diets of 15 atrophic (dry) ARMD patients with an additional portion of five ounces of sauteed spinach four to seven times per week. Study subjects were given a battery of tests to establish baseline measurements of contrast sensitivity, low-luminance, low-contrast visual acuity and glare recovery in each eye. Subsequent measurements were made at intervals ranging from two months to 12 months.

Richer observed improvements in visual function in as little as three months. Often striking improvements in vision were detected through follow-up tests, even when the patient did not report subjective vision changes. Partial or complete resolution of metamorphopsia (distorted vision) and scotomas (blind spots) was reported in seven of eight applicable cases.

Dietary treatment of ARMD should receive more attention, due to its simplicity, low cost and potential application in a broad range of ARMD cases, according to Richer. “There is a preoccupation by clinicians and researchers with exudative (wet) ARMD and a continuing commercial and professional interest in higher-technology, expensive treatment approaches,” said Richer. “Such therapies are applicable to a small percentage of ARMD patients and the disease must be detected early for treatment to be effective.”

Prevent Blindness America, the nation’s leading eye health organisation, estimates 13 million people in the U.S. have symptoms of ARMD, and the disease causes visual impairment in 1.2 million. Richer said this is particularly alarming as the population over the age of 65 is projected to double by the year 2050.

Lutein and Zeaxanthin, related Carotenoids found in leafy green vegetables like spinach and kale, are concentrated in retinal macular pigment and accumulation is dependent on dietary intake.

Macular pigment may filter blue light that damages photoreceptors and the retinal pigment epithelium. In addition, Carotenoids may limit oxidant stress resulting from metabolism and light, acting as antioxidants.

“There is hope for people who suffer from ARMD as well as those at risk of the disease. This disease is preventable, and now may even be treatable with the right dietary modifications or intake of Lutein supplements,” said Richer.

Improved Nutrition Could Help Prevent Vision Loss

BOSTON – “Vision loss associated with aging may be preventable – even reversible – through improved nutrition”, scientists at ‘The Schepens Eye Research Institute‘ said today.

“The traditional thinking is that as age increases, visual sensitivity decreases. But what we are saying is, maybe that’s not inevitable,” said D. Max Snodderly, Ph.D., head of the laboratory at The Schepens where the research was done. “Improved nutrition could help to retard the loss of visual sensitivity with age. Perhaps the gradual loss of vision in many older people is not an inevitable consequence of the aging process.”

In the latest study, Schepens scientists have found that macular pigment in the retina – an accumulation of yellow compounds from fruits and vegetables – may prevent age-related vision loss.

The macula, about the size of a pencil eraser, is at the centre of the retina and is responsible for acute vision. Age-related macular degeneration, the leading cause of blindness in people over age 65 in western industrialised nations, occurs when the aging process destroys the macula.

Snodderly, with co-authors Billy Hammond, a former postdoctoral associate now at Arizona State University, Phoenix, and Billy R. Wooten, Professor of Psychology at Brown University, report in the current issue of Investigative Ophthalmology & Visual Science (Feb. 1998, Vol. 39, No. 2) that macular pigment or dietary factors associated with it appear to protect the retina from loss of sensitivity, when the eye is adapted for light as well as for dark. The National Institutes of Health, the Massachusetts Lions Eye Research Fund, and Kemin Foods funded the research.

Macular pigment is composed of two chemicals, Lutein and Zeaxanthin that are part of the family of compounds known as Carotenoids. These compounds are contained in plants and protect them from light damage. Particularly high concentrations are contained in dark green and orange plants and fruits, like spinach, broccoli, green beans, corn and peaches. (But not carrots. Although carrots are good for Vitamin A deficiency, the retina does not take up betacarotene, which is the main Carotenoid in carrots). “If you eat a lot of fruits and vegetables, you will be getting a lot of Carotenoids,” Snodderly said. “This already fits in with public health recommendations for a wide range of health issues – increase intake of vegetables and fruits.”

The scientists measured macular pigment and visual sensitivity of 27 healthy older subjects, ages 60 to 84, and compared them with 10 younger healthy subjects, ages 24 to 36. Visual sensitivity is how much energy is required to detect photons. Low sensitivity usually associated with aging, means that it is more difficult to see light and thus, there is some vision loss.

The researchers measured macular pigment density (how much light the pigment absorbs) by flashing blue and green lights in an alternate pattern so the lights appeared to flicker. The blue is absorbed by macular pigment, but not green. The amount of blue light required being equally as effective as the green yields the pigment density.

This technology, which could become an important tool in predicting the risk for macular degeneration, is the subject of a recently filed patent application.

As expected, visual sensitivity declined significantly with age. However, macular pigment was a clear determinant of visual sensitivity: Subjects over age 60 with high macular pigment density had the same visual sensitivity as the younger subjects. Older subjects with low macular pigment density had lower visual sensitivity than the younger subjects.

“The results show that high macular pigment density was associated with the retention of youthful visual sensitivity,” the authors write, “which suggested that macular pigment might retard age-related declines in visual function.”

Macular pigment density also appears to be correlated with factors that increase the risk of disease, such as age-related macular degeneration. For example, smoking, female gender and blue irises all are risk factors for the disease, and people with those risk factors also have lower macular pigment density.

Conversely, people with higher macular pigment density are associated with factors that decrease risk, such as high blood concentrations of Carotenoids, high dietary intake of Lutein and Zeaxanthin and dark irises.

“Taken together, the evidence suggests that macular pigment may protect against retinal disease by reducing damage that occurs as we age,”Snodderly said.

Further, macular pigment density may be useful as a predictor for overall ocular health, the researchers said, because macular pigment density correlates with preservation of clarity of the lens as well as sensitivity of the retina. Since previous research by others showed that reduced visual sensitivity may predict development of advanced age-related macular degeneration, then, the researchers suggest, increasing macular pigment density might prevent people who are mildly impaired from advancing to the worst stages of the disease.

They write: “It is possible that, by improving protection, retinal or retinal pigment epithelial cells that may be damaged, but still viable, could recover.”

Said Snodderly, “It appears that people lose visual sensitivity before the worst stages of disease. But with the right nutritional program, you might prevent the low macular pigment group from getting worse, and in the best cases even recover some of the lost function.”

The research also has resulted in a way to tailor dietary recommendations to an individual, the scientists said.

“Our studies have suggested that individuals differ in their ability to absorb nutrients from food into their tissues,” Hammond said. “Thus, some individuals can have relatively high intake of fruits and vegetables and high nutrient blood levels but actually low levels of retinal nutrients. The ability to easily measure macular pigment in vivo may allow us to identify such individuals and determine whether they are at special risk for disease. In the future we may be able to use such technology to tailor our dietary recommendations based on individual responses rather than relying on averaged effects.”

2. Pre-treatment with Alpha Lipoic acid has been found to reduce neuronal damage from excitotoxic damage from cyanide, glutamate and iron ions, demonstrating a strong neuroprotective effect for this substance in nerve tissue. It may be useful in treating glaucoma. (Ed. Note: Recent evidence points to the importance of neuroprotection against glutamate in glaucoma patients, with many medications now heavily promoting this property!) J Cereb Blood Flow Metab 1995 Jul;15(4):624-30 Altern Med Rev 1998 Aug;3(4):308-11 (Other ALA/Neuroprotective Studies)

3. The eyes of open angle glaucoma patients were found to have significantly lower vitamin C levels, as well as higher levels of lipic peroxidation by-products compared to normals. Aleksidze, et al. Oftalmol Zh 1989;(2):114-6

4. Patients suffering from open angle glaucoma and normal-tension glaucoma that were given magnesium twice daily showed improvement of the visual field and reduced peripheral vasospasms (which can cause glaucoma, stroke and heart attack) after four weeks of treatment. Gaspar, et al; Ophthalmologica 1995;209(1):11-3

5. Glaucoma patients treated with vitamin B12 for over 5 years demonstrated better visual acuity and better overall control of their disease. Glacome, 1992; Nippon Ganka Kiyo. 1965 Mar.; Oftalmol Zh. 1965; 20(6); Klin Oczna 1974 Nov;44(11):1183-7

6. Besides beta-carotene, other Carotenoids found in dark green leafy vegetables appear to be much more essential to the health of the eye. Several studies have shown that Lutein and Zeaxanthin supplements may slow vision loss in glaucoma, and in some cases improve eyesight. Science News, Volume 146.

7. Anthocyanosides, compounds found in the herb bilberry, were shown to markedly improve vascular resistance of the capillary wall in the ciliary body of the eye (the source of excess fluid production). Boll. Ocul. 65 789-95, 1986

8. Chronic open angle glaucoma patients had a statistically significant lower thiamine blood level than controls along with poor absorption of that nutrient. Asregadoo, Ann Ophthalmol 1979 Jul;11(7):1095-1100

9. Lipoic acid may be useful in the treatment of glaucoma and may help prevent ischemic optic nerve damage. Altern Med Rev 1998 Aug;3(4):308-11

10. The scientific literature supports recommending Lipoic acid in complex with vitamins B1, B2, B5, B6 (pyridoxal phosphate), and vitamin C to glaucoma patients. Filina, AA & Sporova, NA. Vestn Oftalmol 1991 May-Jun;107(3):19-21

11. Vascular obstruction and hindrance of the blood flow and impaired nutrition of neuronal tissue might be the primary causes of glaucoma. Sonnsjo & Krakau, Acta Ophthalmol (Copenh) 1993 Aug;71(4):433-44

12. Ginkgo Biloba is a potentially important agent in the treatment of glaucoma. It improves central and peripheral blood flow, reduces vasospasm, reduces serum viscosity, has antioxidant activity, platelet activating factor inhibitory activity, and inhibits apoptosis and excitotoxicity – all factors in preventing glaucoma vision loss. (Ed. Note: magnesium also has been shown to decrease peripheral vasospasms, improving blood flow and is neuroprotective, as well.) Ritch R. Med Hypotheses 2000 Feb;54(2):221-35

13. The fatty acid DHA (abundant in fish oil), along with B Complex and vitamin E were shown to be helpful in preventing or delaying vision loss associated with glaucoma. Cellini M, et al., Acta Ophthalmol Scand Suppl 1998;(227):41

14. Ginkgo Biloba extract (40mg, three times daily), increased diastolic blood flow by 23% in the ophthalmic artery in glaucoma patients and may be helpful in protecting the optic nerve from further damage and subsequent visual field loss. Chung HS, et al. J Ocul Pharmacol Ther 1999 Jun;15(3):233-40 (NB: Ginkgo must be used with caution by those taking Coumadin, as it may increase its effect – possibly eliminating the need for it?)

15. Besides Alpha Lipoic Acid, other nutrients that are neuroprotective include vitamin E, Ginkgo Biloba, pycnogenol (from grape seed extract or pine bark), and flavonoids. Kobayashi MS, et al. Free Radic Res 2000 Feb;32(2):115-24

2. Vitamin E, vitamin C, alpha-Lipoic acid, and Taurine appear to offer protection against lens damage caused by low-level radiation. Bantseev, et al. Biochem Mol Biol Int 1997 Sep;42(6):1189-97.

3. Dietary Lutein and cryptoxanthin were associated with 70% lower risk of nuclear cataracts in those under age 65. Lyle, et al. Am J Clin Nutr 1999 Feb;69(2):272-7.

4. Dietary intake of protein, vitamins A, C, E, and carotene, niacin, riboflavin, and thiamine significantly decreased the risk of all cataract types. (Combining a variety of antioxidant nutrients produced the greatest effect.) Cumming RG, et al. Ophthalmology 2000 Mar;107(3):450-6 Leske, et al. Arch Ophthalmol 1991 Feb;109(2):244-51.

5. Vitamin E taken with bilberry extract stopped the progression of senile cortical cataracts in 97% of the eyes of human subjects. Ann Ottalmol Clin Ocul, 1989.

6. Low blood levels of vitamin E were associated with approximately twice the risk of both cortical and nuclear cataracts, compared to median or high levels. Vitale, et al. Epidemiology 1993 May;4(3):195-203

7. Smokers were 2.6 times as likely to develop posterior sub capsular cataracts than nonsmokers. Hankinson, et al. JAMA 1992 Aug 26;268(8):994-8

8. Patients with senile cataracts were found to have significantly lower blood and intraocular levels of the mineral selenium than controls. Karakucuk S, et al. Acta Ophthalmol Scand 1995 Aug;73(4):329-32

9. Alpha Lipoic acid can help prevent cataract formation as well as nerve degeneration and radiation injury. Packer, et al. Free Radic Biol Med 1995 Aug;19(2):227-50

6. Low blood levels of vitamin E were associated with approximately twice the risk of both cortical and nuclear cataracts, compared to median or high levels. Vitale, et al. Epidemiology 1993 May;4(3):195-203

7. Smokers were 2.6 times as likely to develop posterior sub capsular cataracts than nonsmokers. Hankinson, et al. JAMA 1992 Aug 26;268(8):994-8

8. Patients with senile cataracts were found to have significantly lower blood and intraocular levels of the mineral selenium than controls. Karakucuk S, et al. Acta Ophthalmol Scand 1995 Aug;73(4):329-32

9. Alpha Lipoic acid can help prevent cataract formation as well as nerve degeneration and radiation injury. Packer, et al. Free Radic Biol Med 1995 Aug;19(2):227-50

2. Vitamin E, vitamin C, alpha-Lipoic acid, and Taurine appear to offer protection against lens damage caused by low-level radiation. Bantseev, et al. Biochem Mol Biol Int 1997 Sep;42(6):1189-97.

3. Dietary Lutein and cryptoxanthin were associated with 70% lower risk of nuclear cataracts in those under age 65. Lyle, et al. Am J Clin Nutr 1999 Feb;69(2):272-7.

4. Dietary intake of protein, vitamins A, C, E, and carotene, niacin, riboflavin, and thiamine significantly decreased the risk of all cataract types. (Combining a variety of antioxidant nutrients produced the greatest effect.) Cumming RG, et al. Ophthalmology 2000 Mar;107(3):450-6 Leske, et al. Arch Ophthalmol 1991 Feb;109(2):244-51.

5. Vitamin E taken with bilberry extract stopped the progression of senile cortical cataracts in 97% of the eyes of human subjects. Ann Ottalmol Clin Ocul, 1989.

1. Patients with retinitis pigmentosa appear to have faulty cellular uptake of the amino acid Taurine. Disturbed utilisation of vitamin A also appears to play a part in retinitis pigmentosa, and a subgroup of patients benefit from supplementation of this vitamin. Head KA, Altern Med Rev 1999 Oct;4(5):342-59
2. The essential fatty acids alpha-Linolenic acid and DHA may be required for those with retinitis pigmentosa to support normal functional development of the retina. Uauy R, et al. Lipids 1996 Mar;31 Suppl:S167-76
3. A diet high in polyunsaturated fats (e.g. corn oil), and low in vitamin E, selenium, sulphurcontaining amino acids (e.g. Taurine, methionine), and chromium were related to a build up of pigment that mimics one type of retinitis pigmentosa. Katz, et al. Invest Ophthalmol Vis Sci 1978
4. Lutein supplementation in high doses improved both visual acuities and visual fields after 2-4 weeks of treatment in 16 subjects, and was especially effective in blue-eyed individuals. Dagnelie G., et al Optometry 2000 Mar;71(3):147-64

2. Biewenga, G., Haenen G., Bast, A., ‘The pharmacology of the antioxidant Lipoic acid’, Gen. Pharmacol. 1997 Sept; 29(3): 315-31

3. Jacob, S., et al. ‘Enhancement of glucose disposal in patients with type 2 diabetes by Alpha-Lipoic acid’. Arzn.-Forsch, 1995;45:872-4.

4. Ziegler D., Gries, F., ‘Alpha-Lipoic Acid in the treatment of diabetic peripheral and cardiac autonomic neuropathy’, Diabetes, 1997 Sept46 Supp12562.6.

5. Meletis, C., ‘Basic Nutrient Support for Proper Immune Function’. Alternative & Complementary Therapies, Feb. 1999, p.44.

6. Konrad, T., et al ‘Alpha Lipoic Acid treatment decreases serum lactate and pyruvate concentrations and improves glucose effectiveness in lean and obese patients with type 2 diabetes’, Diabetes Care. 1999 Feb; 22(2): 280-7.

7. Whiteman, M., et al. ‘Protection against peroxynitrite-dependent tyrosine nitration and alanti-proteinase inactivation by oxidised and reduced lipoic acid’, FEBS Letters, 1996; 379:74-6.

8. Wickramasinghe, S., Hasan, R, ‘ln Vitro Effects of Vitamin ‘C’, Thioctic Acid and Dihydrolipoic Acid on the Cytotoxicity of Post-Ethanol Serum’, Biochemical Pharmacology, 1992; 43(3): 407-11.

9. Maitra, I, et al ‘Alpha-Lipoic acid prevents buthionine sulfoximine – Induced cataract formation in newborn Rats’, Free Radical Biology & Medicine, 1995-18:823-829.

10. Bustamante, J., et al ‘Antioxidant inhibition of thymocyte apoptosis by Dihydrolipoic acid’, Free Radical Biol. & Med, 1995; 19:339-47.

11. Barbiroli, B., et al. ‘Lipoic (thioctic acid) increases brain energy available and skeletal muscle performance as shown in vivo 31:-MRS in a patient with mitochondrial cytopathy’, J. Neurol., 1995;242:472-7.

12. Schonhit, K, et al. ‘Effect of Alpha-Lipoic Acid and Dihydrolipoic acid on ischemia/reperfuslon injury of the heart and heart mitochondria’, Biochimica et Biophysics Acta., 1995; 1271:335-42.

13. Greenamyre, J., et al. ‘The endogenous cofactors, thioctic acid and Dihydrolipoic acid, are neuroprotective against NMDA and malonlc acid Lesions of striatum’, Neuroscience Letters, 1994; 171:17-20

14. Mitsui, Y, et al ‘Alpha-Lipoic acid provides neuroprotection from ischemla-reperfusion injury of peripheral nerve’, J. Neuro. Sci., Feb. 1; 163(1): 11-6.

15. Stol S., et al. ‘The potent free radical scavenger Alpha Lipoic Acid improves memory in aged mice: putative relationship to NMDA receptor deficits’, Pharmacol, Biochem.& Behavior, 1993: 46:799.805.

2. Consuming 6 mg of the Caroteinoid Lutein (w/ its co-nutrient Zeaxanthin) daily for five months was shown to significantly increase macular pigment density, which protects from harmful blue wavelength light believed to be most responsible for macular degeneration.
Landrum, et al. Exp Eye Res 1997 Jul;65(1):57-62

3. High macular pigment density was associated with the retention of youthful visual sensitivity, which suggested that increasing macular pigment might retard age-related declines in visual function. Hammond, et al. Invest Ophthalmol Vis Sci 1997 Aug; 38(9): 1795-801.

4. Persons who had the lowest serum levels of Lycopene, the most abundant Carotenoid in the serum, were twice as likely to have macular degeneration when compared to those with the highest levels. Mares-Perlman, et al. Arch Ophthalmol 1995 Dec;113(12):1518-23

5. Those consuming Lutein rich foods (spinach and collard greens) five days per week were 8 times less likely to develop macular degeneration as those consuming them once per month. Seddon, et al. JAMA 1994 Nov 9;272(18):1413-20

6. The ARMD population manifested decreased intake of vitamin E, magnesium, zinc, vitamin B6 and folic acid. Patients with advanced ARMD taking antioxidants twice-daily maintained vision in their better functioning eyes significantly better than those taking a placebo. Richer, J Am Optom Assoc 1996 Jan;67(1):12-29 J Am Optom Assoc 1996 Jan;67(1):30-49

7. Smokers with early macular degeneration who consumed the lowest amounts of Carotenoids were nearly 6X as likely to develop advanced macular degeneration than those consuming the highest amounts. Seddon, et al. J. Amer Med Assoc; 1994.

8. In a clinical trial 60% of subjects with ARMD or diabetic macular oedema who received 500 mg of vitamin C, 400 IU of vitamin E, 15,000 IU of beta-carotene and selenium showed either improvement or no further progression of their disease. So Med J, 1987.

9. The evidence suggests that Carotenoids and antioxidant vitamins may help to retard some of the destructive processes in the retina and the retinal pigment epithelium that are responsible for age-related degeneration of the macula. Am J Clin Nutr 1995 Dec;62(6 Suppl):1448S-1461S

10. The minerals copper and zinc are required to synthesize superoxide dismutase and other enzymes in the retina which scavenge free radicals, preventing the oxidative damage which plays a role in the development of drusen, an early sign of Age-Related Macular
Degeneration. Olin, et al: Proc Soc Exp Biol Med 1995 Apr;208(4):370-7

11. Glutathione and its related enzyme precursor amino acids (N-Acetyl-Cysteine, L-Glycine, and glutamine and selenium) are protective against damage to human retinal pigment epithelium cells. Sternberg, Davidson, Jones, et al. Invest Ophthalmol Vis Sci 1993 Dec;34(13):3661-8

12. Quercetin protected bovine retinas in vitro from induced lipid peroxidation, especially when combined with vitamin E, suggesting a potential protective effect in age-related macular degeneration. Ophthalmic Res 1996;28(3):184-92.

13. Deficiency of Taurine, an amino acid, has been shown to lead to retinal degeneration and supplementing it has been used with some success to prevent, treat and stabilise retinal changes. Altern Med Rev 1998 Apr;3(2):128-36. Oftalmol Zh 1989;(8):463-5 Brain Res Brain Res Rev 1991 May-Aug;16(2):151-69 J Neurosci Res 1987;18(4):602-14

14. After 18 months, subjects with macular degeneration who took antioxidants on a consistent basis were two and a half times more likely to improve on visual acuity testing, and four times less likely to deteriorate in their worst eye, compared to those who took them less consistently. Olson, et al. J. Cat Refr Surg, Mar 1991.

15. Patients with confluent soft drusen, or ‘pre-wet’ ARMD, were found to have evidence of vitamin B6 deficiency. B. Lane, Ann Mtg Amer Coll of Nutrition, 1991.

16. General measures for prevention and remediation of macular degeneration would include a combination of supplementation with trace elements, antioxidants and other vitamins, …increasing physical fitness, improving nutrition (e.g. avoiding hydrogenated oils), abstaining from smoking, and protection from excessive light exposure. Eur J Med Res 1997 Oct 30;2(10):445-54

17. There is an association between both low serum selenium levels and current smoking status and the development of age-related macular degeneration. Doc Ophthalmol 1992;81(4):387-400 Mayer, et al. Acta Ophthalmol Scand 1998 Feb;76(1):62-7

18. There was an inverse relationship between dietary pro-vitamin A Carotenoid and vitamin E consumption and the incidence of large macular drusen, as well as between zinc levels and the incidence of retinal pigment abnormalities. Am J Epidemiol 1998 Jul 15;148(2):204-14

19. The evidence suggests that Carotenoids and antioxidant vitamins may help to retard some of the destructive processes in the retina and the retinal pigment epithelium that lead to age-related degeneration of the macula. Snodderly, Am J Clin Nutr 1995;62(6 suppl):1448S-61S

20. Subnormal zinc and/or vitamin E serum levels may be associated with as much as an 82% increased risk of advanced age-related macular degeneration. Vitamin C and Lipoic acid help to recycle vitamin E in the retinal tissues. Ishihara, et al. Nippon Ganka Gakkai Zasshi 1997 Mar;101(3):248-51. Delcourt C. et al. Arch Ophthalmol 1999 Oct;117(10):1384- 90 Stoyanovsky DA, et al. Curr Eye Res 1995 Mar;14(3):181-9

21. In a study of adults over 60 there was found to be a significant link between risk of macular degeneration and low blood levels of vitamin E as well as increased sun exposure. Belda, et al Mech Ageing Dev 1999 Mar 1;107(2):159-64

22. A statistically significant improvement in visual acuity was observed after treatment with Ginkgo Biloba extract in a double blind, placebo controlled study of macular degeneration patients. Lebuisson DA, et al. Presse Med 1986 Sep 25;15(31):1556-8

23. Those consuming fish more than once per week were only half as likely to develop macular degeneration than those consuming it less than once per month. Those consuming the highest amount of cholesterol in their diet were 2.7 times more likely to develop advanced macular degeneration. Smith, et al. Arch Ophthalmol 2000 Mar;118(3):401-4

24. Dietary enzymes increase Glutathione synthesis that can prevent free radical-induced apoptosis (cell suicide) and may help prevent or treat AMD. Progress in Retinal and Eye Research, 2000, Vol. 19, Iss.2 pp 205-221 (No abstract available)

*Absorption Ratesonly in a HEALTHY Digestive tract. Absorption of Lutein available per dose from tablets and Caps may decrease if unhealthy tract. Intra-Oral is not effected by digestive tract health. The Lutein Antioxidant Supplementation Trial (LAST)

The following is a transcript of the abstract to be presented at ARVO 2002 meeting by: S.P. Richer-1, W.Stiles-1, L.Statkute-2, K. Y. Pei-1, J.Frankowski-1, J.Nyland-1, J.Pulido-3, D.Rudy-4.

1. Eye Clinic, DVA Medical Centre, North Chicago, IL, USA
2. Cook County Hospital, Chicago, IL, USA
3. Eye and Ear Infirmary, UIC, Chicago, IL, USA
4. Family Medicine, FUHS/Chicago Medical School, North Chicago, IL, USA.

Main Findings:

Objectives:

Age Related Macular Degeneration (AMD) is the leading cause of loss of vision in ageing western societies, accounting for well over half of visual disability in the United States. The atrophic variety constitutes 90 % of cases and is without an accepted standardised evaluation and treatment protocol. In this abstract the authors describe an intervention treatment study of atrophic AMD using a standardised protocol and nutritional supplementation.

Methods:

The design was a prospective 12-month, placebo controlled, double blind, repeated measures and crossover clinical trial of Lutein vs. Lutein plus antioxidants on 90 mostly male outpatient veterans (74.7 yrs +1- 7.1 yrs) with atrophic (dry form) AMD. A serial-visit clinical evaluation protocol previously described and validated with a pilot case series data (Richer et al, JAOA 1999; 70: 13-23) was used.

Ophthalmic measures were macular pigment optical density (MPOD), lens opacification rating, contrast sensitivity (CSF), low-luminance low-contrast visual acuity, glare recovery (GR) and activities of daily living associated with AMD (night driving / glare adaptation disturbance).

Results:

The three treatment groups (10 mg Lutein, 10 mg Lutein plus antioxidants and placebo) were matched for: age, years diagnosed with AMD, smoking/cardiovascular history, iris colour, lens opacification and nutritional status/physical activity level.

Average eye MPOD, as measured by heterochromic flicker photometry, increased on average by 0.09 log units (repeated factors ANOVA; p<0.05), or 50 % in both the Lutein and Lutein plus antioxidant treatment groups by 12 months. There was statistically significant concurrent improvement in some measures of visual function including GR, CSF and distance/near visual acuity in both treatment groups. Crossover, double crossover and video-documentation of patient symptoms pre and post treatment, were consistent with objective data. Inclusion of multiple nutrients (besides Lutein) appears to provide an added improvement to CSF.

Summary of the main outcome of the LAST relatively to the placebo group.

Conclusions:

Reversibility of AMD symptoms has important biophysical, physiological and clinical implications. This population prospective clinical trial agrees with previous studies suggesting AMD to be a nutrition responsive disorder. The authors believe that low technology visual evaluation protocols, combined with Lutein based nutritional intervention recommendations may improve the eye health of older patients with the dry form of AMD.

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